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Astrocyte Kir4.1 ion channel deficits contribute to neuronal dysfunction in Huntington's disease mod
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日期：2026-04-23
In this study, the authors show that altered medium spiny neuron excitability and symptom onset in Huntington's disease model mice is associated with decreased expression of Kir4.1 and impaired K+ handling by astrocytes. Exogenous expression of Kir4.1 cou...看更多
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